• ‘Too big to fail’ was bad enough for the banks. Now we have ‘too many to fail.’
    Last Updated: Feb. 13, 2024 at 1:20 p.m. ET

    People line up outside of the shuttered Silicon Valley Bank headquarters on March 10, 2023, in Santa Clara, Calif.
    Getty Images
    Almost a year after the mini banking crisis in the United States, it is worth revisiting the episode. Was it just a tempest in a teacup? Was there really a systemic threat, or was it just a problem with a few banks? Should the interventions by the U.S. Federal Reserve and Treasury worry or comfort us?

    Recall that three mid-size U.S. banks suddenly failed around March 2023. The most prominent was Silicon Valley Bank, which became the second-largest bank failure in U.S. history, after Washington Mutual in 2008. Roughly 90% of the deposits at SVB were uninsured, and uninsured deposits are prone to runs. Making matters worse, SVB had invested significant sums in long-term bonds, the market value of which fell as interest rates rose. When SVB sold some of these holdings to raise funds, the unrealized losses embedded in its bond portfolio started coming to light. A failed equity offering then triggered a classic bank run.

    It is convenient to think that these issues were confined to just a few rogue banks. But the problem was systemic.

    When the Fed engages in quantitative easing (QE), it buys bonds from financial institutions. Typically, those sellers then deposit the money in their bank, and this results in a large increase in uninsured deposits in the banking system. On the banks’ asset side, there is a corresponding increase in central-bank reserves. This is stable, since reserves are the most liquid asset on the planet and can be used to satisfy any impatient depositors who come for their money. Unfortunately, a number of smaller banks (with less than $50 billion in assets) moved away from this stable position as QE continued.

    Historically, smaller U.S. banks financed themselves conservatively, with uninsured demandable deposits accounting for only around 10% of their liabilities. Yet by the time the Fed was done with its pandemic-era QE, these banks’ uninsured demandable deposits exceeded 30% of liabilities. Though that level was still far below SVB’s, these institutions clearly had drunk from the same firehose.

    Smaller banks were also more conservative about liquidity in the past. At the outset of QE in late 2008, banks with less than $50 billion in assets had reserves (and other assets that could be used to borrow reserves) that exceeded the uninsured demandable deposits they had issued. By early 2023, however, they had issued runnable claims (in aggregate) that were one and a half times the size of their liquid assets. Instead of holding liquid reserves, their assets were now more weighted toward long-term securities and term lending, including a significant share of commercial real-estate (CRE) loans.

    Advertisement
    Thus, as the Fed raised interest rates, the economic value of these banks’ assets fell sharply. Some of the fall was hidden by accounting sleight of hand, but SVB’s sudden demise caused investors to scrutinize banks’ balance sheets more carefully. What they saw did not instill confidence. The KBW Nasdaq Bank Index duly fell by over 25%, and deposits started flowing out of a large number of banks, many of which lacked the liquidity to accommodate the sudden outflows. The risk of contagious runs across smaller banks was real, as was the possibly of the problem spreading more widely.

    The Treasury essentially took bank runs off the table, while the Fed provided banks the funds to accommodate the continuing — though no longer panicked — depositor outflows.

    Importantly, as private money flowed to large banks, very little flowed to small- and medium-size institutions. That is why the authorities had to come to the rescue. Soon after SVB’s demise, the Treasury signaled that no uninsured depositor in small banks would suffer losses in any further bank collapses.

    The Fed opened a generous new facility that lent money for up to one year to banks against the par, or face value, of the securities they held on their balance sheets, without adjusting for the erosion in the value of these securities from higher interest rates. And the Federal Home Loan Banks (FHLBanks) — effectively an arm of the U.S. government — increased its lending to stressed banks, with total advances to the banking system having already tripled between March 2022 and March 2023 amid the Fed’s policy tightening. Borrowing by small- and medium-size banks from these official sources skyrocketed.

    The Treasury essentially took bank runs off the table, while the Fed provided banks the funds to accommodate the continuing — though no longer panicked — depositor outflows. A potential banking crisis was converted into a slow-burning problem for banks as they recognized and absorbed the losses on their balance sheets.

    Just recently, New York Community Bancorp NYCB, -5.17%, which bought parts of one of the banks that failed in 2023, reminded us that this process is still underway when it announced large losses. With the Russell microcap index of small companies significantly underperforming the S&P 100 index OEX of the largest companies since March 2023, it appears that smaller banks’ troubles have weighed on their traditional clients: small- and medium-size companies.

    Where does that leave us? Although the situation could have been much worse if the Treasury and the Fed had not stepped in, the seeming ease with which the panic was arrested allowed public attention to move on. Apart from die-hard libertarians, no one seems to care much about the extent of the intervention that was needed to rescue the smaller banks, nor has there been any broad inquiry into the circumstances that led to the vulnerabilities.

    As a result, several questions remain unanswered. To what extent were the seeds of the 2023 banking stress sown by the pandemic-induced monetary stimulus and lax supervision of what banks did with the money? Did advances by the FHLBanks delay failed banks’ efforts to raise capital? Are banks that relied on official backstops after SVB’s failure keeping afloat distressed CRE borrowers, and therefore merely postponing an eventual reckoning?

    It is not good for capitalism when those who knowingly take risks — bankers and uninsured depositors, in this case — pay no price when a risk materializes. Despite sweeping banking reforms over the past 15 years, the authorities have once again shown that they are willing to bail out market players if enough of them have taken the same risk.

    “Too big to fail” was bad enough, but now we have “too many to fail.” The mini-crisis of March 2023 was much more than a footnote in banking history. We cannot afford to bury it.

    Raghuram G. Rajan, a former governor of the Reserve Bank of India, is professor of finance at the University of Chicago Booth School of Business and the author, most recently, of Monetary Policy and Its Unintended Consequences (The MIT Press, 2023). Viral V. Acharya, a former deputy governor of the Reserve Bank of India, is professor of economics at New York University’s Stern School of Business.

    This commentary was published with the permission of Project Syndicate — The Danger of Forgetting the 2023 Banking Crisis.

    More: Regional-bank bondholders seem unworried by New York Community Bank’s problems

    Also read: Recession in 2024? A quarter of economists think it will happen.


    😎🇺🇸🦅 PAR-TY… 🎉. https://www.marketwatch.com/story/too-big-to-fail-was-bad-enough-for-the-banks-now-we-have-too-many-to-fail-d89dcdda
    ‘Too big to fail’ was bad enough for the banks. Now we have ‘too many to fail.’ Last Updated: Feb. 13, 2024 at 1:20 p.m. ET People line up outside of the shuttered Silicon Valley Bank headquarters on March 10, 2023, in Santa Clara, Calif. Getty Images Almost a year after the mini banking crisis in the United States, it is worth revisiting the episode. Was it just a tempest in a teacup? Was there really a systemic threat, or was it just a problem with a few banks? Should the interventions by the U.S. Federal Reserve and Treasury worry or comfort us? Recall that three mid-size U.S. banks suddenly failed around March 2023. The most prominent was Silicon Valley Bank, which became the second-largest bank failure in U.S. history, after Washington Mutual in 2008. Roughly 90% of the deposits at SVB were uninsured, and uninsured deposits are prone to runs. Making matters worse, SVB had invested significant sums in long-term bonds, the market value of which fell as interest rates rose. When SVB sold some of these holdings to raise funds, the unrealized losses embedded in its bond portfolio started coming to light. A failed equity offering then triggered a classic bank run. It is convenient to think that these issues were confined to just a few rogue banks. But the problem was systemic. When the Fed engages in quantitative easing (QE), it buys bonds from financial institutions. Typically, those sellers then deposit the money in their bank, and this results in a large increase in uninsured deposits in the banking system. On the banks’ asset side, there is a corresponding increase in central-bank reserves. This is stable, since reserves are the most liquid asset on the planet and can be used to satisfy any impatient depositors who come for their money. Unfortunately, a number of smaller banks (with less than $50 billion in assets) moved away from this stable position as QE continued. Historically, smaller U.S. banks financed themselves conservatively, with uninsured demandable deposits accounting for only around 10% of their liabilities. Yet by the time the Fed was done with its pandemic-era QE, these banks’ uninsured demandable deposits exceeded 30% of liabilities. Though that level was still far below SVB’s, these institutions clearly had drunk from the same firehose. Smaller banks were also more conservative about liquidity in the past. At the outset of QE in late 2008, banks with less than $50 billion in assets had reserves (and other assets that could be used to borrow reserves) that exceeded the uninsured demandable deposits they had issued. By early 2023, however, they had issued runnable claims (in aggregate) that were one and a half times the size of their liquid assets. Instead of holding liquid reserves, their assets were now more weighted toward long-term securities and term lending, including a significant share of commercial real-estate (CRE) loans. Advertisement Thus, as the Fed raised interest rates, the economic value of these banks’ assets fell sharply. Some of the fall was hidden by accounting sleight of hand, but SVB’s sudden demise caused investors to scrutinize banks’ balance sheets more carefully. What they saw did not instill confidence. The KBW Nasdaq Bank Index duly fell by over 25%, and deposits started flowing out of a large number of banks, many of which lacked the liquidity to accommodate the sudden outflows. The risk of contagious runs across smaller banks was real, as was the possibly of the problem spreading more widely. The Treasury essentially took bank runs off the table, while the Fed provided banks the funds to accommodate the continuing — though no longer panicked — depositor outflows. Importantly, as private money flowed to large banks, very little flowed to small- and medium-size institutions. That is why the authorities had to come to the rescue. Soon after SVB’s demise, the Treasury signaled that no uninsured depositor in small banks would suffer losses in any further bank collapses. The Fed opened a generous new facility that lent money for up to one year to banks against the par, or face value, of the securities they held on their balance sheets, without adjusting for the erosion in the value of these securities from higher interest rates. And the Federal Home Loan Banks (FHLBanks) — effectively an arm of the U.S. government — increased its lending to stressed banks, with total advances to the banking system having already tripled between March 2022 and March 2023 amid the Fed’s policy tightening. Borrowing by small- and medium-size banks from these official sources skyrocketed. The Treasury essentially took bank runs off the table, while the Fed provided banks the funds to accommodate the continuing — though no longer panicked — depositor outflows. A potential banking crisis was converted into a slow-burning problem for banks as they recognized and absorbed the losses on their balance sheets. Just recently, New York Community Bancorp NYCB, -5.17%, which bought parts of one of the banks that failed in 2023, reminded us that this process is still underway when it announced large losses. With the Russell microcap index of small companies significantly underperforming the S&P 100 index OEX of the largest companies since March 2023, it appears that smaller banks’ troubles have weighed on their traditional clients: small- and medium-size companies. Where does that leave us? Although the situation could have been much worse if the Treasury and the Fed had not stepped in, the seeming ease with which the panic was arrested allowed public attention to move on. Apart from die-hard libertarians, no one seems to care much about the extent of the intervention that was needed to rescue the smaller banks, nor has there been any broad inquiry into the circumstances that led to the vulnerabilities. As a result, several questions remain unanswered. To what extent were the seeds of the 2023 banking stress sown by the pandemic-induced monetary stimulus and lax supervision of what banks did with the money? Did advances by the FHLBanks delay failed banks’ efforts to raise capital? Are banks that relied on official backstops after SVB’s failure keeping afloat distressed CRE borrowers, and therefore merely postponing an eventual reckoning? It is not good for capitalism when those who knowingly take risks — bankers and uninsured depositors, in this case — pay no price when a risk materializes. Despite sweeping banking reforms over the past 15 years, the authorities have once again shown that they are willing to bail out market players if enough of them have taken the same risk. “Too big to fail” was bad enough, but now we have “too many to fail.” The mini-crisis of March 2023 was much more than a footnote in banking history. We cannot afford to bury it. Raghuram G. Rajan, a former governor of the Reserve Bank of India, is professor of finance at the University of Chicago Booth School of Business and the author, most recently, of Monetary Policy and Its Unintended Consequences (The MIT Press, 2023). Viral V. Acharya, a former deputy governor of the Reserve Bank of India, is professor of economics at New York University’s Stern School of Business. This commentary was published with the permission of Project Syndicate — The Danger of Forgetting the 2023 Banking Crisis. More: Regional-bank bondholders seem unworried by New York Community Bank’s problems Also read: Recession in 2024? A quarter of economists think it will happen. 😎🇺🇸🦅 PAR-TY… 🎉. https://www.marketwatch.com/story/too-big-to-fail-was-bad-enough-for-the-banks-now-we-have-too-many-to-fail-d89dcdda
    WWW.MARKETWATCH.COM
    ‘Too big to fail’ was bad enough for the banks. Now we have ‘too many to fail.’
    The failures may have been confined to just a few rogue banks, but the problem is systemic.
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  • Can 2 Cheap Meds, 1 Vitamin & Baking Soda Kill Any Cancer?
    Ivermectin, Fenbendazole, Vit C and Sodium Bicarb. But don't worry your cancer is safe because the FDA would never allow it.

    Dr. Syed Haider
    Cancer Treatment Options | Houston Methodist
    Cancer rates have skyrocketed in the past century for a number of reasons not least of which is the incredibly large number of toxins spewed into the environment and incorporated into our food supplies. And now with most of humanity exposed to the cancerous spike protein there is likely to be even further acceleration. Those exposed to the fallout from the East Palestine Ohio train wreck, which may spread quite widely along the eastern seaboard, are particularly at risk of developing cancer in the coming months and years from the ingition of the vinyl chloride cargo and it’s toxic breakdown products, especially dioxins.

    This post is not meant to be an exhaustive treatise on the prevention and treatment of cancer, but only to explain as simply as possible the scientific theory behind Adam Gaertner’s anti-cancer protocol, which combines 4 simple and cheap therapies that have been separately used and studied for a wide variety of human cancers with mixed results, but together have powerful synergistic effects that may, it is hoped, effectively eliminate any cancer. And at the end his simple 3 week protocol is included.

    Before we begin I also have to say that I have seen many people beat end stage cancer using drastic elimination diets and a modifed Gerson juicing protocol. And of course I have known many who decided on chemotherapy, radiation and surgery. Both paths are extremely difficult and require a lot of commitment and sacrifice. Perhaps the following protocol can help more people more easily overcome cancer.

    And after cancer is beaten, it pays to address the root causes because those who overcome cancer are often prone to an even more aggressive recurrence, especially if they persist in the unhealthy exposures and lifestyle habits that triggered it in the first place.

    WHAT IS CANCER?

    All tissues are made up of individual cellular building blocks that work together to accomplish a joint function. For example liver cells are like millions of workmen that all together make up the liver. Normally tissues maintain just the right amount of helpful worker cells. As old cells die off, new ones take their place.

    Cancers arise from cells in normal tissues that start to grow uncontrollably - the old workmen don't want to die and instead find a way to become immortal. They also don't want to work anymore and begin using up resources like the nutrients and oxygen coming into the tissue via the blood. These immortal cells also multiply very quickly and if left unchecked can destroy the normal cells and then the entire organ ceases to function. Not only that but they also enter the bloodstream and travel to other distant organs and take up new residence and continue to multiply out of control.


    Just as there are a tiny percentage of psychopaths and criminals in every society, who attempt to murder others and appropriate all the resources for themselves, there are cancer cells in everyone's bodies all the time that would like nothing better than to take over.

    Thank you for reading Dr. Syed Haider. This post is public so feel free to share it.

    Share

    And just as nations utilize a police force and military to maintain the peace, our bodies utilize specialized immune system processes and immune cells to keep the cancer cells in check - to continuously search them out and put them to death.

    However, when these defenses fail due to exposure to various carcinogens or simply old age, cancerous cells can gain a foothold and destroy us.

    DEFENSES AGAINST CANCER

    Intracellular Cytosolic Immunity

    Think of a cell like a 3D sphere. Inside the sphere there is another smaller sphere, which is the nucleus and holds the genetic material or DNA. Everything outside the nucleus is called the cytoplasm.

    Steph's Nature and Science
    Each individual cell has an internal immune system, called the cytosolic immune system that will monitor the cells health, and if the cell becomes cancerous will kill it in a process of cellular suicide termed apoptosis.

    You can imagine this as a person's conscience.

    Think of a horror movie scenario where someone becomes bitten by a mindless zombie and begins to change into a zombie themselves, feeling the first stirrings of hunger for the blood of those around them. Knowing they are doomed and wanting to preserve the lives of their loved ones they commit suicide rather than becoming a monster.

    In this way our own first line of defense against cancer is a system of internal checks and balances that will lead to cellular suicide or apoptosis.

    The checks and balances are a system of pro-suicide (pro-apoptotic) and anti-suicide (anti-apoptotic) pathways: p53 tumor suppressor gene, G1/S checkpoint, Hippo, TGF-β, Wnt signaling, Notch signaling, and PI3K/AKT signaling.

    Within these extremely complex pathways made up of numerous interacting chemical messengers there are just a small handful of signals that can lead to cellular death: caspases, apoptosis inducing factor (AIF), endonucleases, granzymes, BH3-interacting domain death agonist (Bid), Death receptor 5 (DR5), Fas-associated protein with death domain (FADD).

    A vast majority of cancers arise due to mutations affecting these critical cytosolic immunity pathways.

    So the conscience of the cell, its own internal checks and balances, become distorted and do not trigger suicide as they should when the cell begins transforming into a cancer cell.

    2 Zombie Stocks Coming Back from the Dead | Nasdaq
    The mutations work by producing malformed proteins that do not do their usual job of triggering cellular suicide.

    Usually malformed proteins would themselves be destroyed by the intracellular “chaperone” and “proteasome” systems - these are both meant to protect our cells from mutations.

    The reason this does not happen in the case of most cancers is that most cancers also stimulate an internal process that makes them more resistant to the chaperone and proteasome systems - by way of the production of heat shock protein 90 (hsp90).

    Ivermectin

    Doctors Sue FDA For Prohibiting Use Of Ivermectin To Treat Covid
    Ivermectin, the horse and cow and human drug, has traditionally been used as an antiparasitic (e.g. scabies), but also has antiviral and anti-inflammatory activities. It binds to hsp90 and other heat shock proteins blocking their ability to stabilize mutated checkpoint proteins. It likewise suppresses a number of the anti-apoptotic pathway especially TGF-β, as well as increasing the expression of p53 tumor suppressor gene pro-apoptotic pathway.

    So in effect ivermectin helps the cancer cell reestablish the ability to detect that it is cancerous and thereby trigger an internal process of suicide.

    Unfortunately not every cancer utilizes the pathways ivermectin targets.

    And as a result of the relatively rapid replication rate of cancerous cells, and the evolutionary imperative to survive, additional mutations are often present across the tumor mass. As a result, ivermectin may be effective against only 90% of a given tumor mass; however, if the 90% is killed in this way, the remaining 10% will, by default, not be able to be corrected, leading to relapse, with the remainder becoming harder to treat - as the 10% left over multiplies and becomes the entire 100% of tumor.

    Extracellular Natural Killer Cell Immunity

    Immense Immunology Insight: Girl, if we were lymphocytes... You'd be a ...
    Another arm of the immune system that protects against cancer is outside the cancer cell itself. We can think of this like the police force that keeps an eye out for dangerous cancer cells.

    Our internal police force uses markers to identify healthy cells and unhealthy cells as well as foreign intruders like bacteria and viruses.

    The markers our immune system uses for identification are called antigens - little bits of cells.

    Most of our immune cells are trained to recognize foreign particles that do not belong and destroy them - like crazy immigration agent death squads.

    But the Natural Killer (NK) cells are trained to check for what is supposed to be present - self-antigens - markers that indicate normal cells, kind of like ID cards.

    In policing terms: NK cells wander the streets and demand everyone's papers, regardless of any evidence of a crime, and immediately execute anyone who cannot prove they belong.

    "Ihre Papiere, bitte!" (Episode 48) | #FSCK 'Em All!
    The rapid rate of replication of cancerous cells places them under heavy evolutionary pressure; those cells that do not express self-antigens will be targeted and destroyed by the NK cells, whereas those that do may not be - so some cancer cells develop the ability to forge their own papers and pass themselves off as normal law abiding residents, rather than dangerous alien invaders.

    Those wily ones will multiply while the others die off, and eventually the entire tumor mass is comprised of cells that can trick the NK cells into leaving them alone by presenting proper identification, even though they will still be presenting other signs of being foreign - like devil horns growing out of their heads - “it’s just part of my mardi gras outfit officer”.

    While this is very bad news it does open up an avenue of treatment via T cell activation.

    T cell Immunity

    CD 4 T cells are also called helper T cells, they aid other immune cells via the release of cytokine messengers. CD 8 T cells are also called cytotoxic T cells. Cyto for cell, toxic for toxic - i.e. they kill cancer cells.

    T cells like NK cells detect self antigens and will ignore those that present them, but they also look for non self antigens (like those devil horns) as well as an additional costimulatory signal to trigger their death squad role.

    It’s like they not only check your papers, but they check to make sure those horns are actually real and they make you pass a lie detector test. If they find real horns and sense signs of stress during the lie detector test they have enough evidence to declare you guilty and execute you.

    Geek Comic for November 17th - You can Beat the Lie Detector Test Because…
    If they just find the horns, but no signs of stress, they let you go on your way.

    Cancer cells can’t avoid making weird mutated horn-like proteins, but they can figure out how to pass the lie detector test by muting their stress signals.

    The way to bypass that is by subjecting them to so much stress that their ability to mute the signs of stress breaks down, and at the same time triggering more foreign proteins and stopping proliferation would also be helpful, which brings us to the other 3 therapies.

    Fenbendazole, Sodium Bicarbonate & Vitamin C

    Fenbendazole

    Panacur Granules 22.2% [Fenbendazole] (1 lb)
    Humans are not listed on the side panel
    Fenbendazole is not FDA approved for use in humans, but is commonly used as an antiparasitic medication in animals, and has been studied in some human cancer studies, where it appears to be safe. It has multiple effects against cancer cells. Most significantly, it can lead to the influence the MAPK pathway to activate cellular suicide or apoptosis.

    It destabilizes cellular protein structures called microtubules that are essential to cell division.

    It also disrupts cancer cell energy production by blocking the breakdown of sugar (glycolysis) which is like crude oil for cells and also blocking the ability of mitochondria, the energy refining factories of cells from using the crude oil to produce the cellular equivalent of electricity, i.e. ATP - the universal bioenergy molecule.

    This collection of actions may not be applicable for all cancers, however a sizable proportion are affected; as such metabolic disruption occurs which then leads to production of cellular stress signals.

    An important manifestation of this is CD80, a costimulatory signal that in combination with T Cell Receptor binding to a foreign antigen, activates CD8 T-cells; alternatively if the antigen is self, it will inhibit them, as well as activate dormant NK cells in the area.

    Share

    So what’s happening here is if the cancer cell has non self antigens (those devil horns) the stress signals (failed lie detector test) will activate CD8 cytotoxic T cells to kill it.

    If however the cancer cell shows a normal self antigen to the T cell along with the stress signals, the T cell will stand down but the same stress signals may still activate nearby NK cells.

    Thereby some of the tumor cells will be destroyed releasing many new antigens into the area, both self and non self. These new antigens will be recognized by nearby immune cells and train them to better detect the remaining tumor cells. This triggers a far more robust immune activation and ends up in effectively nuking the area - destroying all remaining tumor as well as some friendlies and innocent bystanders mixed up in the fray.

    Sodium Bicarbonate

    Alkaline Diet for Cancer : Comprehensive Nutrional Guide to Cure and ...
    The mechanism of sodium bicarbonate action is easy to understand, based on the Warburg effect: decreasing acidity (increasing the pH or alkalinity) outside the cancer cells impairs their ability to maintain a highly alkaline environment within themselves. That alters cancer cells' metabolism, prompting similar immune system reactions as previously discussed and igniting further cascades.

    Unfortunately, if sodium bicarbonate is used without other agents from the protocol, tumors promptly become resistant and cancer-fighting benefits decrease to mere prolongation of life expectancy instead of complete elimination.

    Vitamin C

    Best Linus Pauling Cancer Vitamin C - Your Best Life
    When ascorbic acid is used in large quantities, along with the reduced form dehydroascorbate (DHA), it induces intense oxidative stress within cancerous cells; if that stress is insufficient to destroy the cell outright, it triggers the release of numerous cytokines, including our friend CD80, which initiates the cascade described above involving CD8 cytotoxic T cells.

    Not all forms of cancer are responsive to this pathway and sodium bicarbonate is capable of directly counteracting it.

    As a potent immunomodulator vitamin C even has the potential to disrupt the inflammatory response involved in targeting a significant-sized tumor.

    So it’s important to carefully balance the two options, and not use both simultaneously. The alkalization brought about by sodium bicarbonate won't last for particularly long; therefore, employing one after another in alternating fashion will likely provide more benefits than using just one of them at a time.

    In a Nutshell

    The following are four therapeutic pathways that, when used together, cause cancerous cells to undergo both apoptosis and loss of immune evasion features so the immune system can identify and attack them.

    Ivermectin inhibits mutant checkpoint and cascade transduction proteins, particularly PI3K, reduces TAM anti-apoptotic signaling, and increases expression of the tumor suppressor p53 by binding to the hsp90 protein.

    In addition to modulating the MAPK pathway, fenbendazole destabilizes microtubules, inhibits glycolytic metabolism, inhibits mitochondrial oxidative phosphorylation, and reduces anti-apoptotic PD-L1 expression feedback loops.

    Through alkalization of the cytosolic tumor environment, sodium bicarbonate induces metabolic stress.

    Vitamin C triggers oxidative stress and cytokine production.

    In this method, cytosolic apoptosis signaling cascades are promoted, and effector CD8 and NK cells are infiltrated into a tumor mass through adaptive recognition of foreign antigens and inhibition of anti-apoptotic pathways in order to achieve complete remission through both self-destruct signaling pathways as well as inflammatory immune destruction of cancerous cells.

    The Proposed Protocol

    Unlike most traditional cytotoxic cancer therapies that destroy both cancer cells as well as regular cells and especially the body's immune system cells, this protocol stimulates the body's own innate and adaptive immune system to fight off cancer.

    NLRP3 and STING enhance immune attack on cancer | Cancer Biology
    This protocol should not be used in combination with most mainstream cancer treatments, such as chemotherapy or radiotherapy, due to their ability to impair the immune system that the protocol depends on.

    It is likely to be most potent at the early stages of disease; further progress of the condition will prolong duration of treatment needed.

    A healthy immune system takes time to ramp up the necessary response, so the protocol is based on the time required for each drug to take effect, safety data, bioavailability, and elimination time.

    Day 1:

    Ivermectin: 1 mg/kg by mouth

    Fenbendazole: 1000mg by mouth

    Sodium Bicarbonate: 1 tsp morning and evening dissolved in 1 quart of water

    Day 2:

    Ascorbic acid: 50 mg/kg by mouth, two doses, 8 hours apart or 20g IV, once

    Day 3:

    Repeat Day 1

    Day 4:

    Repeat Day 2

    Days 5 to 10:

    Fenbendazole, 200mg by mouth daily

    Alternate sodium bicarbonate and ascorbic acid every other day beginning with sodium bicarb on day 5, then vitamin C on day 6, etc.

    Day 11:

    Ivermectin: 1 mg/kg by mouth

    Fenbendazole: 1000 mg by mouth

    Sodium Bicarbonate: 1 tsp morning and evening dissolved in 1 quart of water

    Days 12 to 20:

    Sodium Bicarbonate: 1 tsp morning and evening dissolved in 1 quart of water

    Day 20:

    Imaging: Check progress. Significant reduction or complete elimination of tumor mass should have occurred by this time, if not repeat the protocol.

    At this time the US FDA has not approved this protocol for study or for use in humans.

    It is unlikely that any pharmaceutical company will spend the millions of dollars it would take to prove this protocol in large randomized controlled trials because none of the four therapeutics are under patent and therefore cannot be effectively monetized.

    Even if some billionaire decided to back this protocol, Big Pharma would move heaven and earth to prove it doesn’t work as they did with ivermectin and hydroxychloroquine for COVID.

    Let me know below if you know of anyone who has utilized these 4 therapeutics together.

    And finally beating cancer inside us is a great first step to healing our world, but next we need to beat the cancerous psychopaths who are destroying our societies. If not we will go the way of Rome and a new civilization will rise from our ashes.


    I believe in the Judeo Christian ethic of working hard and giving back without big government. My online clinic, mygotodoc.com, exemplifies that by charging a fee that is well worth the service, but also offering free medical answers and (asynchronous) care for anyone that needs it.

    The same applies at my free online Summit Long COVID Reset, exclusive weekly content, including live Q&As and much more released on my video subscription platform, and in my course, Phoenix for Healing Long Haul and Lean Vitality - all are available for a fee or for free by request.

    So thank you to everyone who finds this written content valuable and supports it by being a paid subscriber (even though there are currently no paid subscriber benefits aside from a warm fuzzy feeling that you did something good). You are helping enable the significant amount of time and effort it takes to write. If you have the means also please consider donating to help support the care of those cannot afford it at mygotodoc.com/donation.

    If you are a free subscriber thanks for being here, and please also consider supporting my efforts in any way you can, but especially by sharing my posts widely.

    https://blog.mygotodoc.com/p/can-2-cheap-meds-1-vitamin-and-baking
    Can 2 Cheap Meds, 1 Vitamin & Baking Soda Kill Any Cancer? Ivermectin, Fenbendazole, Vit C and Sodium Bicarb. But don't worry your cancer is safe because the FDA would never allow it. Dr. Syed Haider Cancer Treatment Options | Houston Methodist Cancer rates have skyrocketed in the past century for a number of reasons not least of which is the incredibly large number of toxins spewed into the environment and incorporated into our food supplies. And now with most of humanity exposed to the cancerous spike protein there is likely to be even further acceleration. Those exposed to the fallout from the East Palestine Ohio train wreck, which may spread quite widely along the eastern seaboard, are particularly at risk of developing cancer in the coming months and years from the ingition of the vinyl chloride cargo and it’s toxic breakdown products, especially dioxins. This post is not meant to be an exhaustive treatise on the prevention and treatment of cancer, but only to explain as simply as possible the scientific theory behind Adam Gaertner’s anti-cancer protocol, which combines 4 simple and cheap therapies that have been separately used and studied for a wide variety of human cancers with mixed results, but together have powerful synergistic effects that may, it is hoped, effectively eliminate any cancer. And at the end his simple 3 week protocol is included. Before we begin I also have to say that I have seen many people beat end stage cancer using drastic elimination diets and a modifed Gerson juicing protocol. And of course I have known many who decided on chemotherapy, radiation and surgery. Both paths are extremely difficult and require a lot of commitment and sacrifice. Perhaps the following protocol can help more people more easily overcome cancer. And after cancer is beaten, it pays to address the root causes because those who overcome cancer are often prone to an even more aggressive recurrence, especially if they persist in the unhealthy exposures and lifestyle habits that triggered it in the first place. WHAT IS CANCER? All tissues are made up of individual cellular building blocks that work together to accomplish a joint function. For example liver cells are like millions of workmen that all together make up the liver. Normally tissues maintain just the right amount of helpful worker cells. As old cells die off, new ones take their place. Cancers arise from cells in normal tissues that start to grow uncontrollably - the old workmen don't want to die and instead find a way to become immortal. They also don't want to work anymore and begin using up resources like the nutrients and oxygen coming into the tissue via the blood. These immortal cells also multiply very quickly and if left unchecked can destroy the normal cells and then the entire organ ceases to function. Not only that but they also enter the bloodstream and travel to other distant organs and take up new residence and continue to multiply out of control. Just as there are a tiny percentage of psychopaths and criminals in every society, who attempt to murder others and appropriate all the resources for themselves, there are cancer cells in everyone's bodies all the time that would like nothing better than to take over. Thank you for reading Dr. Syed Haider. This post is public so feel free to share it. Share And just as nations utilize a police force and military to maintain the peace, our bodies utilize specialized immune system processes and immune cells to keep the cancer cells in check - to continuously search them out and put them to death. However, when these defenses fail due to exposure to various carcinogens or simply old age, cancerous cells can gain a foothold and destroy us. DEFENSES AGAINST CANCER Intracellular Cytosolic Immunity Think of a cell like a 3D sphere. Inside the sphere there is another smaller sphere, which is the nucleus and holds the genetic material or DNA. Everything outside the nucleus is called the cytoplasm. Steph's Nature and Science Each individual cell has an internal immune system, called the cytosolic immune system that will monitor the cells health, and if the cell becomes cancerous will kill it in a process of cellular suicide termed apoptosis. You can imagine this as a person's conscience. Think of a horror movie scenario where someone becomes bitten by a mindless zombie and begins to change into a zombie themselves, feeling the first stirrings of hunger for the blood of those around them. Knowing they are doomed and wanting to preserve the lives of their loved ones they commit suicide rather than becoming a monster. In this way our own first line of defense against cancer is a system of internal checks and balances that will lead to cellular suicide or apoptosis. The checks and balances are a system of pro-suicide (pro-apoptotic) and anti-suicide (anti-apoptotic) pathways: p53 tumor suppressor gene, G1/S checkpoint, Hippo, TGF-β, Wnt signaling, Notch signaling, and PI3K/AKT signaling. Within these extremely complex pathways made up of numerous interacting chemical messengers there are just a small handful of signals that can lead to cellular death: caspases, apoptosis inducing factor (AIF), endonucleases, granzymes, BH3-interacting domain death agonist (Bid), Death receptor 5 (DR5), Fas-associated protein with death domain (FADD). A vast majority of cancers arise due to mutations affecting these critical cytosolic immunity pathways. So the conscience of the cell, its own internal checks and balances, become distorted and do not trigger suicide as they should when the cell begins transforming into a cancer cell. 2 Zombie Stocks Coming Back from the Dead | Nasdaq The mutations work by producing malformed proteins that do not do their usual job of triggering cellular suicide. Usually malformed proteins would themselves be destroyed by the intracellular “chaperone” and “proteasome” systems - these are both meant to protect our cells from mutations. The reason this does not happen in the case of most cancers is that most cancers also stimulate an internal process that makes them more resistant to the chaperone and proteasome systems - by way of the production of heat shock protein 90 (hsp90). Ivermectin Doctors Sue FDA For Prohibiting Use Of Ivermectin To Treat Covid Ivermectin, the horse and cow and human drug, has traditionally been used as an antiparasitic (e.g. scabies), but also has antiviral and anti-inflammatory activities. It binds to hsp90 and other heat shock proteins blocking their ability to stabilize mutated checkpoint proteins. It likewise suppresses a number of the anti-apoptotic pathway especially TGF-β, as well as increasing the expression of p53 tumor suppressor gene pro-apoptotic pathway. So in effect ivermectin helps the cancer cell reestablish the ability to detect that it is cancerous and thereby trigger an internal process of suicide. Unfortunately not every cancer utilizes the pathways ivermectin targets. And as a result of the relatively rapid replication rate of cancerous cells, and the evolutionary imperative to survive, additional mutations are often present across the tumor mass. As a result, ivermectin may be effective against only 90% of a given tumor mass; however, if the 90% is killed in this way, the remaining 10% will, by default, not be able to be corrected, leading to relapse, with the remainder becoming harder to treat - as the 10% left over multiplies and becomes the entire 100% of tumor. Extracellular Natural Killer Cell Immunity Immense Immunology Insight: Girl, if we were lymphocytes... You'd be a ... Another arm of the immune system that protects against cancer is outside the cancer cell itself. We can think of this like the police force that keeps an eye out for dangerous cancer cells. Our internal police force uses markers to identify healthy cells and unhealthy cells as well as foreign intruders like bacteria and viruses. The markers our immune system uses for identification are called antigens - little bits of cells. Most of our immune cells are trained to recognize foreign particles that do not belong and destroy them - like crazy immigration agent death squads. But the Natural Killer (NK) cells are trained to check for what is supposed to be present - self-antigens - markers that indicate normal cells, kind of like ID cards. In policing terms: NK cells wander the streets and demand everyone's papers, regardless of any evidence of a crime, and immediately execute anyone who cannot prove they belong. "Ihre Papiere, bitte!" (Episode 48) | #FSCK 'Em All! The rapid rate of replication of cancerous cells places them under heavy evolutionary pressure; those cells that do not express self-antigens will be targeted and destroyed by the NK cells, whereas those that do may not be - so some cancer cells develop the ability to forge their own papers and pass themselves off as normal law abiding residents, rather than dangerous alien invaders. Those wily ones will multiply while the others die off, and eventually the entire tumor mass is comprised of cells that can trick the NK cells into leaving them alone by presenting proper identification, even though they will still be presenting other signs of being foreign - like devil horns growing out of their heads - “it’s just part of my mardi gras outfit officer”. While this is very bad news it does open up an avenue of treatment via T cell activation. T cell Immunity CD 4 T cells are also called helper T cells, they aid other immune cells via the release of cytokine messengers. CD 8 T cells are also called cytotoxic T cells. Cyto for cell, toxic for toxic - i.e. they kill cancer cells. T cells like NK cells detect self antigens and will ignore those that present them, but they also look for non self antigens (like those devil horns) as well as an additional costimulatory signal to trigger their death squad role. It’s like they not only check your papers, but they check to make sure those horns are actually real and they make you pass a lie detector test. If they find real horns and sense signs of stress during the lie detector test they have enough evidence to declare you guilty and execute you. Geek Comic for November 17th - You can Beat the Lie Detector Test Because… If they just find the horns, but no signs of stress, they let you go on your way. Cancer cells can’t avoid making weird mutated horn-like proteins, but they can figure out how to pass the lie detector test by muting their stress signals. The way to bypass that is by subjecting them to so much stress that their ability to mute the signs of stress breaks down, and at the same time triggering more foreign proteins and stopping proliferation would also be helpful, which brings us to the other 3 therapies. Fenbendazole, Sodium Bicarbonate & Vitamin C Fenbendazole Panacur Granules 22.2% [Fenbendazole] (1 lb) Humans are not listed on the side panel Fenbendazole is not FDA approved for use in humans, but is commonly used as an antiparasitic medication in animals, and has been studied in some human cancer studies, where it appears to be safe. It has multiple effects against cancer cells. Most significantly, it can lead to the influence the MAPK pathway to activate cellular suicide or apoptosis. It destabilizes cellular protein structures called microtubules that are essential to cell division. It also disrupts cancer cell energy production by blocking the breakdown of sugar (glycolysis) which is like crude oil for cells and also blocking the ability of mitochondria, the energy refining factories of cells from using the crude oil to produce the cellular equivalent of electricity, i.e. ATP - the universal bioenergy molecule. This collection of actions may not be applicable for all cancers, however a sizable proportion are affected; as such metabolic disruption occurs which then leads to production of cellular stress signals. An important manifestation of this is CD80, a costimulatory signal that in combination with T Cell Receptor binding to a foreign antigen, activates CD8 T-cells; alternatively if the antigen is self, it will inhibit them, as well as activate dormant NK cells in the area. Share So what’s happening here is if the cancer cell has non self antigens (those devil horns) the stress signals (failed lie detector test) will activate CD8 cytotoxic T cells to kill it. If however the cancer cell shows a normal self antigen to the T cell along with the stress signals, the T cell will stand down but the same stress signals may still activate nearby NK cells. Thereby some of the tumor cells will be destroyed releasing many new antigens into the area, both self and non self. These new antigens will be recognized by nearby immune cells and train them to better detect the remaining tumor cells. This triggers a far more robust immune activation and ends up in effectively nuking the area - destroying all remaining tumor as well as some friendlies and innocent bystanders mixed up in the fray. Sodium Bicarbonate Alkaline Diet for Cancer : Comprehensive Nutrional Guide to Cure and ... The mechanism of sodium bicarbonate action is easy to understand, based on the Warburg effect: decreasing acidity (increasing the pH or alkalinity) outside the cancer cells impairs their ability to maintain a highly alkaline environment within themselves. That alters cancer cells' metabolism, prompting similar immune system reactions as previously discussed and igniting further cascades. Unfortunately, if sodium bicarbonate is used without other agents from the protocol, tumors promptly become resistant and cancer-fighting benefits decrease to mere prolongation of life expectancy instead of complete elimination. Vitamin C Best Linus Pauling Cancer Vitamin C - Your Best Life When ascorbic acid is used in large quantities, along with the reduced form dehydroascorbate (DHA), it induces intense oxidative stress within cancerous cells; if that stress is insufficient to destroy the cell outright, it triggers the release of numerous cytokines, including our friend CD80, which initiates the cascade described above involving CD8 cytotoxic T cells. Not all forms of cancer are responsive to this pathway and sodium bicarbonate is capable of directly counteracting it. As a potent immunomodulator vitamin C even has the potential to disrupt the inflammatory response involved in targeting a significant-sized tumor. So it’s important to carefully balance the two options, and not use both simultaneously. The alkalization brought about by sodium bicarbonate won't last for particularly long; therefore, employing one after another in alternating fashion will likely provide more benefits than using just one of them at a time. In a Nutshell The following are four therapeutic pathways that, when used together, cause cancerous cells to undergo both apoptosis and loss of immune evasion features so the immune system can identify and attack them. Ivermectin inhibits mutant checkpoint and cascade transduction proteins, particularly PI3K, reduces TAM anti-apoptotic signaling, and increases expression of the tumor suppressor p53 by binding to the hsp90 protein. In addition to modulating the MAPK pathway, fenbendazole destabilizes microtubules, inhibits glycolytic metabolism, inhibits mitochondrial oxidative phosphorylation, and reduces anti-apoptotic PD-L1 expression feedback loops. Through alkalization of the cytosolic tumor environment, sodium bicarbonate induces metabolic stress. Vitamin C triggers oxidative stress and cytokine production. In this method, cytosolic apoptosis signaling cascades are promoted, and effector CD8 and NK cells are infiltrated into a tumor mass through adaptive recognition of foreign antigens and inhibition of anti-apoptotic pathways in order to achieve complete remission through both self-destruct signaling pathways as well as inflammatory immune destruction of cancerous cells. The Proposed Protocol Unlike most traditional cytotoxic cancer therapies that destroy both cancer cells as well as regular cells and especially the body's immune system cells, this protocol stimulates the body's own innate and adaptive immune system to fight off cancer. NLRP3 and STING enhance immune attack on cancer | Cancer Biology This protocol should not be used in combination with most mainstream cancer treatments, such as chemotherapy or radiotherapy, due to their ability to impair the immune system that the protocol depends on. It is likely to be most potent at the early stages of disease; further progress of the condition will prolong duration of treatment needed. A healthy immune system takes time to ramp up the necessary response, so the protocol is based on the time required for each drug to take effect, safety data, bioavailability, and elimination time. Day 1: Ivermectin: 1 mg/kg by mouth Fenbendazole: 1000mg by mouth Sodium Bicarbonate: 1 tsp morning and evening dissolved in 1 quart of water Day 2: Ascorbic acid: 50 mg/kg by mouth, two doses, 8 hours apart or 20g IV, once Day 3: Repeat Day 1 Day 4: Repeat Day 2 Days 5 to 10: Fenbendazole, 200mg by mouth daily Alternate sodium bicarbonate and ascorbic acid every other day beginning with sodium bicarb on day 5, then vitamin C on day 6, etc. Day 11: Ivermectin: 1 mg/kg by mouth Fenbendazole: 1000 mg by mouth Sodium Bicarbonate: 1 tsp morning and evening dissolved in 1 quart of water Days 12 to 20: Sodium Bicarbonate: 1 tsp morning and evening dissolved in 1 quart of water Day 20: Imaging: Check progress. Significant reduction or complete elimination of tumor mass should have occurred by this time, if not repeat the protocol. At this time the US FDA has not approved this protocol for study or for use in humans. It is unlikely that any pharmaceutical company will spend the millions of dollars it would take to prove this protocol in large randomized controlled trials because none of the four therapeutics are under patent and therefore cannot be effectively monetized. Even if some billionaire decided to back this protocol, Big Pharma would move heaven and earth to prove it doesn’t work as they did with ivermectin and hydroxychloroquine for COVID. Let me know below if you know of anyone who has utilized these 4 therapeutics together. And finally beating cancer inside us is a great first step to healing our world, but next we need to beat the cancerous psychopaths who are destroying our societies. If not we will go the way of Rome and a new civilization will rise from our ashes. I believe in the Judeo Christian ethic of working hard and giving back without big government. My online clinic, mygotodoc.com, exemplifies that by charging a fee that is well worth the service, but also offering free medical answers and (asynchronous) care for anyone that needs it. The same applies at my free online Summit Long COVID Reset, exclusive weekly content, including live Q&As and much more released on my video subscription platform, and in my course, Phoenix for Healing Long Haul and Lean Vitality - all are available for a fee or for free by request. So thank you to everyone who finds this written content valuable and supports it by being a paid subscriber (even though there are currently no paid subscriber benefits aside from a warm fuzzy feeling that you did something good). You are helping enable the significant amount of time and effort it takes to write. If you have the means also please consider donating to help support the care of those cannot afford it at mygotodoc.com/donation. If you are a free subscriber thanks for being here, and please also consider supporting my efforts in any way you can, but especially by sharing my posts widely. https://blog.mygotodoc.com/p/can-2-cheap-meds-1-vitamin-and-baking
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    Can 2 Cheap Meds, 1 Vitamin & Baking Soda Kill Any Cancer?
    Ivermectin, Fenbendazole, Vit C and Sodium Bicarb. But don't worry your cancer is safe because the FDA would never allow it.
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  • 🚨🗞️#Bitcoin #mining company #GRIID, an #American-based firm, is poised to resume trading on the #Nasdaq stock exchange.
    🚨🗞️#Bitcoin #mining company #GRIID, an #American-based firm, is poised to resume trading on the #Nasdaq stock exchange.
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  • #1. My all time favorite and largest hold is SME, and with good reason. SoMee is one of the most underrated decetralized social media platforms out there. SoMee keeps their Community Updated Daily, Consistently and often in their Telegram channel here: https://t.me/Someeofficials

    The SoMee Devs are top of the line Devs like i haven't seen in many other platforms. The Devs are squashing bugs and adding features to the site daily. They have German Actors and Actresses in the Platform, along with Influencers and famous people. They are and also have been featured in the Nasdaq,

    Source: https://youtu.be/4cTfRImSOhQ
    https://youtu.be/x1hIntXParc
    https://youtu.be/hFJ13GPxZhs

    Further Sources: https://youtu.be/Tf1YENqydJ8
    https://youtu.be/pIHWY17P1ZU
    https://youtu.be/S8XNi0WJy90

    #2. PoB: Yes, this project really called my attention because of its similarities to Bitcoin, Limited supply at 21 million like Bitcoin and a halving every four years of rewards very similar to Bitcoin. I love the PoB platform which i have enjoyed even more than Leofinance as it is super user friendly, and you can stake tokens from many project through their platform. I have close to 11K PoB now and am looking to continue accumulating at these prices.

    #3. MemeHive: I am very thankful to the MemeHive Community for teaching me the proper way of Mememing by sourcing images and Memes properly. For this i have accumulated close to 1 Million Meme tokens. From using their layer 2, i noticed that they have a very close relationship with the Lolz currency. The future looks bright for something like this, coupled with Pepe and other Meme coins on the Hive Network! Much props to that team.

    #4. Leofinance: Although i was really enjoying this platform. Lately it is falling from grace, at least as a Social Media Platform for me. I love their Dex and strongly believe it is the best and most comprehensive DEX i have ever used. In fact, nothing comes even remotely close to their DEX. Their. Token is taking off yes, and i bet there are good reasons for that.

    #5. Pepe Token: Yes, i have posted about this one before. I will cover this one much much more, as more and more features are implemented. This one has the potential of being far superior than the Ethereum version regardless of whether it makes you rich in the short term or not.

    #6. Archon: I really intended to buy a lot more of this one but was disappointed that i could not find any platform at all for this. I can't view anything on this, and i can't even use their app on Hive Keychain nor Hivesigner. Hence, i cannot test anything. The only place i can find to stake this oddly enough was on the PoB Web-App. I was going to get 15K of this one for now but changed my mind because i am not really happy with this one at the moment. I tried posting with their tribe tag in order to get answers, and for nine hours no one answered. So, i deleted my post...

    Source of Image: My Account's Stats
    ![](https://images.ecency.com/DQmb7x2y5MPKGdTGLz11vhNU7az8XmYFWi6CHkWT6VNxomX/screenshot_20230517_171433_1_.png)
    #1. My all time favorite and largest hold is SME, and with good reason. SoMee is one of the most underrated decetralized social media platforms out there. SoMee keeps their Community Updated Daily, Consistently and often in their Telegram channel here: https://t.me/Someeofficials The SoMee Devs are top of the line Devs like i haven't seen in many other platforms. The Devs are squashing bugs and adding features to the site daily. They have German Actors and Actresses in the Platform, along with Influencers and famous people. They are and also have been featured in the Nasdaq, Source: https://youtu.be/4cTfRImSOhQ https://youtu.be/x1hIntXParc https://youtu.be/hFJ13GPxZhs Further Sources: https://youtu.be/Tf1YENqydJ8 https://youtu.be/pIHWY17P1ZU https://youtu.be/S8XNi0WJy90 #2. PoB: Yes, this project really called my attention because of its similarities to Bitcoin, Limited supply at 21 million like Bitcoin and a halving every four years of rewards very similar to Bitcoin. I love the PoB platform which i have enjoyed even more than Leofinance as it is super user friendly, and you can stake tokens from many project through their platform. I have close to 11K PoB now and am looking to continue accumulating at these prices. #3. MemeHive: I am very thankful to the MemeHive Community for teaching me the proper way of Mememing by sourcing images and Memes properly. For this i have accumulated close to 1 Million Meme tokens. From using their layer 2, i noticed that they have a very close relationship with the Lolz currency. The future looks bright for something like this, coupled with Pepe and other Meme coins on the Hive Network! Much props to that team. #4. Leofinance: Although i was really enjoying this platform. Lately it is falling from grace, at least as a Social Media Platform for me. I love their Dex and strongly believe it is the best and most comprehensive DEX i have ever used. In fact, nothing comes even remotely close to their DEX. Their. Token is taking off yes, and i bet there are good reasons for that. #5. Pepe Token: Yes, i have posted about this one before. I will cover this one much much more, as more and more features are implemented. This one has the potential of being far superior than the Ethereum version regardless of whether it makes you rich in the short term or not. #6. Archon: I really intended to buy a lot more of this one but was disappointed that i could not find any platform at all for this. I can't view anything on this, and i can't even use their app on Hive Keychain nor Hivesigner. Hence, i cannot test anything. The only place i can find to stake this oddly enough was on the PoB Web-App. I was going to get 15K of this one for now but changed my mind because i am not really happy with this one at the moment. I tried posting with their tribe tag in order to get answers, and for nine hours no one answered. So, i deleted my post... Source of Image: My Account's Stats ![](https://images.ecency.com/DQmb7x2y5MPKGdTGLz11vhNU7az8XmYFWi6CHkWT6VNxomX/screenshot_20230517_171433_1_.png)
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  • NASDAQ-listed MicroStrategy, the business intelligence giant founded by Michael Saylor who is also the largest corporate holder of bitcoin, made another notable purchase of $29.3 million of the crypto asset. This helped the company round up its massive BTC stash to 140,000.

    #SoMee #SME #AweSomee #Crypto

    ????(https://cointimes.com.br/microstrategy-quer-levantar-us-500-milhoes-para-comprar-bitcoin/)
    NASDAQ-listed MicroStrategy, the business intelligence giant founded by Michael Saylor who is also the largest corporate holder of bitcoin, made another notable purchase of $29.3 million of the crypto asset. This helped the company round up its massive BTC stash to 140,000. #SoMee #SME #AweSomee #Crypto ????(https://cointimes.com.br/microstrategy-quer-levantar-us-500-milhoes-para-comprar-bitcoin/)
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  • ALTCOIN LOOKING POSITIVE ACROSS THE BOARD

    Bitcoin's dominance is going up but we want to see Bitcoin dominance comes down and the total market go up and this is when Altcoins season will take place.

    Bitcoin dominance is still persisting and Bitcoin is very persisting and Bitcoin is very much leading this. If you look at the last bull market, Bitcoin very much led the charge and all coins played this game of catch-up. Bitcoin leads the charge for the same reason we have seen GBTC go up. You have smart money and where the smart money goes is into Bitcoin, where there is liquidity for it.

    I think Altcoin is looking very positive across the board, I think it will drop down into some of these altcoins into some of the smaller time frames. Much of your altcoins are still under the one wave, coming out of the two-wave correction. Just about to get started with the third wave and we know what the signal for the full set on Altcoin is, it is for Bitcoin dominance to come down. We still got people calling for new lows, your emotions are not going to help you anyway in the market and you need to have a plan.

    I think the macro is very pivoting, I think this is QE but the FED is not saying that. They are determined not to use the word bailout except it is exactly bailout. Interestingly, NASDAQ to offers Bitcoin and crypto trading to block, it thought crypto is getting banned and nobody wants to use it. I must say this is an opportunity of a lifetime right in front of you, still with it and I think you will do well.

    #SME #SoMee #AweSME #crypto #altcoins #someeofficial
    ALTCOIN LOOKING POSITIVE ACROSS THE BOARD Bitcoin's dominance is going up but we want to see Bitcoin dominance comes down and the total market go up and this is when Altcoins season will take place. Bitcoin dominance is still persisting and Bitcoin is very persisting and Bitcoin is very much leading this. If you look at the last bull market, Bitcoin very much led the charge and all coins played this game of catch-up. Bitcoin leads the charge for the same reason we have seen GBTC go up. You have smart money and where the smart money goes is into Bitcoin, where there is liquidity for it. I think Altcoin is looking very positive across the board, I think it will drop down into some of these altcoins into some of the smaller time frames. Much of your altcoins are still under the one wave, coming out of the two-wave correction. Just about to get started with the third wave and we know what the signal for the full set on Altcoin is, it is for Bitcoin dominance to come down. We still got people calling for new lows, your emotions are not going to help you anyway in the market and you need to have a plan. I think the macro is very pivoting, I think this is QE but the FED is not saying that. They are determined not to use the word bailout except it is exactly bailout. Interestingly, NASDAQ to offers Bitcoin and crypto trading to block, it thought crypto is getting banned and nobody wants to use it. I must say this is an opportunity of a lifetime right in front of you, still with it and I think you will do well. #SME #SoMee #AweSME #crypto #altcoins #someeofficial
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  • Nasdaq will launch its own cryptocurrency custody service, and plans to do so by the end of the first half. With this, Nasdaq will join BNY Mellon and Fidelity, among other large companies, that offer custody of Bitcoin and cryptocurrencies.
    Nasdaq will launch its own cryptocurrency custody service, and plans to do so by the end of the first half. With this, Nasdaq will join BNY Mellon and Fidelity, among other large companies, that offer custody of Bitcoin and cryptocurrencies.
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  • Visiting the NASDAQ was awesome! I can’t wait to be back
    Visiting the NASDAQ was awesome! I can’t wait to be back
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  • Great memories with CEO of Fetch.ai $FET Humayun at the NASDAQ in NYC.
    Great memories with CEO of Fetch.ai $FET Humayun at the NASDAQ in NYC.
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  • 6% Rates Incoming? Gundlach Spars With Dimon 24 Hours Before Crucial CPI Print


    Stocks rallied kind of hard, but not really hard yesterday. The Dow added 189 pts or 0.6%, the S&P gained 28 or 0.7%, the Nasdaq advanced by 106 pts or 1%, the Russell added 27 pts or 1.5% while the Transports tacked on 37 pts or 0.3%.

    And it’s the “Fed will cave” story all over again. Bloomberg summed up the day best with the headline: “Stocks Bounce Back with Brewing Optimism Over CPI (https://ca.finance.yahoo.com/news/asia-stocks-set-tepid-opening-230147384.html)”. Meanwhile, two of the biggest names on Wall Street are at odds with one another over where the Federal Reserve is going to head from here.

    After all, brewing optimism is all the smart logic algos needed. Traders and algos raised the temperature in the room yesterday, betting that the forthcoming CPI report will not only weaken, but weaken beyond what the very optimistic estimates already are - thus ‘building a case’ for the Fed to stop the madness and pause further rate hikes beyond March.

    At the end of the day, Doubleline’s CEO Jeffrey Gundlach stood up and said: ‘Don’t be ridiculous, the Fed? Investors need to pay 100% attention to what the bond market is saying rather than what the FED is telling you they are saying.’ In fact, he said it this way: “My 40 plus years of experience in finance strongly recommends that investors should look at what the market says over what the Fed says.”

    This echoes the comments Gundlach (https://twitter.com/TruthGundlach) made on Twitter last week when he said: “There is no way the Fed is going to 5%. The Fed is not in control, the bond market is in control.”

    Meanwhile Yahoo Finance reported yesterday that Wallstreet heavyweight,CEO of JP Morgan, "Jamie Dimon Says Fed 'May Very Well' Raise Interest Rates to 6%"

    (https://finance.yahoo.com/news/jamie-dimon-says-fed-may-very-well-raise-interest-rates-to-6-195147027.html)"Jamie Dimon expects the Federal Reserve will raise interest rates higher than most officials and Wall Street strategists have forecast as the U.S. central bank continues its fight against persistent inflation.

    The chief executive officer of JPMorgan (JPM), the largest consumer bank in the U.S. by assets, said Tuesday in an interview with Fox Business Network that the Fed’s terminal rate may hit 6%, a level notably above the 5% many have called for.

    'Whether 5% interest rates are enough to slow inflation to where it needs to be, I don’t know,' Dimon said during the discussion at JPMorgan’s annual health-care investment-banking conference in San Francisco, citing fiscal stimulus that was 'so large and still largely unspent.'

    'Is it 5%? My view is, it may very well be 6%,' he added.

    This time last year, Dimon was among the first voices on Wall Street to predict – correctly – that Federal Reserve officials would deliver as many as six or seven increases to their benchmark policy rate as prices rose at a historic pace. He said the three or four hikes investors were bracing for at the time were a low estimate.

    In 2022, the U.S. central bank lifted rates seven times to a cumulative increase of 4.25% to the highest in 15 years from near-zero levels. At least 75 basis points more of hikes are expected this year."

    :link:Source: Zerohedge (https://www.zerohedge.com/news/2023-01-11/6-rates-incoming),
    Quoth The Raven (https://quoththeraven.substack.com/p/6-rates-incoming-gundlach-spars-with),
    Yahoo Finance (https://finance.yahoo.com/news/jamie-dimon-says-fed-may-very-well-raise-interest-rates-to-6-195147027.html),
    Bloomberg (https://ca.finance.yahoo.com/news/asia-stocks-set-tepid-opening-230147384.html)

    ???? Follow:
    t.me/g3news
    6% Rates Incoming? Gundlach Spars With Dimon 24 Hours Before Crucial CPI Print Stocks rallied kind of hard, but not really hard yesterday. The Dow added 189 pts or 0.6%, the S&P gained 28 or 0.7%, the Nasdaq advanced by 106 pts or 1%, the Russell added 27 pts or 1.5% while the Transports tacked on 37 pts or 0.3%. And it’s the “Fed will cave” story all over again. Bloomberg summed up the day best with the headline: “Stocks Bounce Back with Brewing Optimism Over CPI (https://ca.finance.yahoo.com/news/asia-stocks-set-tepid-opening-230147384.html)”. Meanwhile, two of the biggest names on Wall Street are at odds with one another over where the Federal Reserve is going to head from here. After all, brewing optimism is all the smart logic algos needed. Traders and algos raised the temperature in the room yesterday, betting that the forthcoming CPI report will not only weaken, but weaken beyond what the very optimistic estimates already are - thus ‘building a case’ for the Fed to stop the madness and pause further rate hikes beyond March. At the end of the day, Doubleline’s CEO Jeffrey Gundlach stood up and said: ‘Don’t be ridiculous, the Fed? Investors need to pay 100% attention to what the bond market is saying rather than what the FED is telling you they are saying.’ In fact, he said it this way: “My 40 plus years of experience in finance strongly recommends that investors should look at what the market says over what the Fed says.” This echoes the comments Gundlach (https://twitter.com/TruthGundlach) made on Twitter last week when he said: “There is no way the Fed is going to 5%. The Fed is not in control, the bond market is in control.” Meanwhile Yahoo Finance reported yesterday that Wallstreet heavyweight,CEO of JP Morgan, "Jamie Dimon Says Fed 'May Very Well' Raise Interest Rates to 6%" (https://finance.yahoo.com/news/jamie-dimon-says-fed-may-very-well-raise-interest-rates-to-6-195147027.html)"Jamie Dimon expects the Federal Reserve will raise interest rates higher than most officials and Wall Street strategists have forecast as the U.S. central bank continues its fight against persistent inflation. The chief executive officer of JPMorgan (JPM), the largest consumer bank in the U.S. by assets, said Tuesday in an interview with Fox Business Network that the Fed’s terminal rate may hit 6%, a level notably above the 5% many have called for. 'Whether 5% interest rates are enough to slow inflation to where it needs to be, I don’t know,' Dimon said during the discussion at JPMorgan’s annual health-care investment-banking conference in San Francisco, citing fiscal stimulus that was 'so large and still largely unspent.' 'Is it 5%? My view is, it may very well be 6%,' he added. This time last year, Dimon was among the first voices on Wall Street to predict – correctly – that Federal Reserve officials would deliver as many as six or seven increases to their benchmark policy rate as prices rose at a historic pace. He said the three or four hikes investors were bracing for at the time were a low estimate. In 2022, the U.S. central bank lifted rates seven times to a cumulative increase of 4.25% to the highest in 15 years from near-zero levels. At least 75 basis points more of hikes are expected this year." :link:Source: Zerohedge (https://www.zerohedge.com/news/2023-01-11/6-rates-incoming), Quoth The Raven (https://quoththeraven.substack.com/p/6-rates-incoming-gundlach-spars-with), Yahoo Finance (https://finance.yahoo.com/news/jamie-dimon-says-fed-may-very-well-raise-interest-rates-to-6-195147027.html), Bloomberg (https://ca.finance.yahoo.com/news/asia-stocks-set-tepid-opening-230147384.html) ???? Follow: t.me/g3news
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  • "Bitcoin has greatly outperformed both the S&P 500 and NASDAQ over the last 10 years but haters continue to call Bitcoin 'worthless'. "

    https://seekingalpha.com/article/4568319-microstrategy-ignore-haters-hold-through-crypto-winter?mailingid=30183429&messageid=cryptodigest&serial=30183429.4494&utm_campaign=nl-cryptodigest&utm_content=cryptodigest&utm_medium=email&utm_source=seeking_alpha&utm_term=30183429.4494

    #someeofficial
    "Bitcoin has greatly outperformed both the S&P 500 and NASDAQ over the last 10 years but haters continue to call Bitcoin 'worthless'. " https://seekingalpha.com/article/4568319-microstrategy-ignore-haters-hold-through-crypto-winter?mailingid=30183429&messageid=cryptodigest&serial=30183429.4494&utm_campaign=nl-cryptodigest&utm_content=cryptodigest&utm_medium=email&utm_source=seeking_alpha&utm_term=30183429.4494 #someeofficial
    SEEKINGALPHA.COM
    MicroStrategy: Ignore Haters And Hold Through The Crypto Winter
    MicroStrategy holds 132,500 BTC, despite selling 780 BTC in Dec. 2022 to carry back capital losses. Find out why Bitcoin bulls should be bullish on MSTR stock.
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  • First Wave Bio NASDAQ (FWBI) is a extremely high risk high return biotech company. I just added to my position here near all time lows.
    First Wave Bio NASDAQ (FWBI) is a extremely high risk high return biotech company. I just added to my position here near all time lows.
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  • Nasdaq launches a digital assets unit to offer #Bitcoin and #Ethereum custody services to institutional investors ????????????
    @WatcherGuru
    Nasdaq launches a digital assets unit to offer #Bitcoin and #Ethereum custody services to institutional investors ???????????? @WatcherGuru
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  • The Nasdaq just posted its worst month since 2008.
    The Nasdaq just posted its worst month since 2008.
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